Proposed pathways underlying diDCP-LA-PE-induced GLUT4 translocation
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Function and dysfunction of aPKC isoforms for glucose transport in insulin-sensitive and insulin-resistant states
Schematic of insulin-induced translocation of glucose transporter 4
LNK deficiency decreases obesity-induced insulin resistance by regulating GLUT4 through the PI3K-Akt-AS160 pathway in adipose tissue - Figure f6
Insulin Signaling: GLUT4 Vesicles Exit via the Exocyst - ScienceDirect
5′-AMP-activated protein kinase increases glucose uptake independent of GLUT4 translocation in cardiac myocytes
The phosphatidylethanolamine derivative diDCP-LA-PE mimics intracellular insulin signaling
The GLUT4 Glucose Transporter - ScienceDirect
Procyanidin Promotes Translocation of Glucose Transporter 4 in Muscle of Mice through Activation of Insulin and AMPK Signaling Pathways
Simplified representation of molecular mechanism involved in insulin
Frontiers Neferine Promotes GLUT4 Expression and Fusion With the Plasma Membrane to Induce Glucose Uptake in L6 Cells
Frontiers Tmod3 Phosphorylation Mediates AMPK-Dependent GLUT4 Plasma Membrane Insertion in Myoblasts
